|
Rodent studies |
||
| Study |
Induction of sepsis |
Acute tubular necrosis |
|
|
||
| Hurley and colleagues [32] |
Salmonella enteritidis endotoxin |
No |
| Miyaji and colleagues [33] |
Cecal ligation perforation/lipopolysaccharide |
Yes |
| Sato and colleagues [34] |
Escherichia coli |
No |
| Hayashi and colleagues [35] |
Lipopolysaccharide-induced sepsis |
No |
| Tsao and colleagues [36] |
Lipopolysaccharide-induced sepsis |
No |
| Kadkhodaee and Qasemi [37] |
Lipopolysaccharide-induced sepsis |
Yes |
| Zager and Prior [38] |
E. coli septicemia |
No |
| Wang and colleagues [39] |
Lipopolysaccharide-induced sepsis |
Yes |
| Tiwari and colleagues [40] |
Lipopolysaccharide-induced sepsis |
No |
| Gallos and colleagues [41] |
Cecal ligation perforation |
No |
| Kikeri and colleagues [42] |
Lipopolysaccharide-induced sepsis |
No |
| Yokota and colleagues [43] |
Lipopolysaccharide-induced sepsis |
No |
Langenberg et al. Critical Care 2008 12:R38 doi:10.1186/cc6823 |
||