Table 3 |
||
|
Rodent studies |
||
|
Study |
Induction of sepsis |
Acute tubular necrosis |
|
|
||
|
Hurley and colleagues [32] |
Salmonella enteritidis endotoxin |
No |
|
Miyaji and colleagues [33] |
Cecal ligation perforation/lipopolysaccharide |
Yes |
|
Sato and colleagues [34] |
Escherichia coli |
No |
|
Hayashi and colleagues [35] |
Lipopolysaccharide-induced sepsis |
No |
|
Tsao and colleagues [36] |
Lipopolysaccharide-induced sepsis |
No |
|
Kadkhodaee and Qasemi [37] |
Lipopolysaccharide-induced sepsis |
Yes |
|
Zager and Prior [38] |
E. coli septicemia |
No |
|
Wang and colleagues [39] |
Lipopolysaccharide-induced sepsis |
Yes |
|
Tiwari and colleagues [40] |
Lipopolysaccharide-induced sepsis |
No |
|
Gallos and colleagues [41] |
Cecal ligation perforation |
No |
|
Kikeri and colleagues [42] |
Lipopolysaccharide-induced sepsis |
No |
|
Yokota and colleagues [43] |
Lipopolysaccharide-induced sepsis |
No |
|
|
||
|
Langenberg et al. Critical Care 2008 12:R38 doi:10.1186/cc6823 |
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